Post-Infarction Inflammation and Left Ventricular Remodeling

non-specific response to tissue damage.1 However, an investigation into the relation between the peak body temperature during the first week and in-hospital complications since the event of MI showed that the incidence of complications of pump failure (grade of class 2 or greater according to Killip’s classification or subset II or greater according to Forrester’s classification) and ventricular aneurysm, as well as the incidence of cardiovascular events such as cardiac death, pump failure, fatal ventricular arrhythmias and cardiac rupture, increased in proportion to the body temperature increase.2 Although the peak creatine kinase (CK) level, an indicator of infarct size, was similar, LV end-diastolic and LV end-systolic volumes measured by left ventriculography before discharge had increased and the LV ejection fraction decreased in proportion to the peak body temperature, demonstrating an association between body temperature increase after MI and LV remodeling.2